All this can affect your healthy weight loss since you may not have the energy to commit 100% to your exercise for caloric burning workouts.
So what influences how much ATP your mitochondria create? Your genetics, your exercise level, your nutritional status, your surroundings of electro smog, or an endocrine problem. Of course you can’t change your genetics, but you dictate and control your exercise, your nutritional status, and how you control your electromagnetic surroundings.
So let’s talk about your mitochondria. As you age, the number of mitochondria in your cells decreases in efficiency by about 5-8 percent every ten years. That explains why you have less energy the older you get. Two ways to reverse this is by 1) exercise and 2) by accurate pulsating electro-magnetic fields such as through the MRS 2000+. Those who exercise or use the MRS 2000+ have more mitochondria, which mean more potential for energy and the ability to burn more calories for healthy weight loss.
Another factor that contributes to low ATP production is oxidative damage to the mitochondria. A deficiency of antioxidants increases this rate of oxidative damage to the mitochondria. Again your nutritional status and exercise contributes to this.
Due to changes in our food intake, eating patterns, and food production methods, many people are eating low iodine rich foods. This correlates to an under functioning thyroid gland. Those who suffer from this have a 60 percent reduction in ATP production.
Make a resolution right now to make sure you are providing the best opportunity for your body to produce ATP and optimize your body to assist with healthy weight loss.
References:
Short KR, Bigelow ML, Kahl J, et al. Decline in skeletal muscle mitochondrial function with aging in humans. Proc Natl Acad Sci USA. 2005; 102: 5618-5623
Carmichael, J. Magnetic resonance stimulation, using the field to maximize your health. 2009; 52-55
Mesnhikova EF, Ritov VB, Fairfull L, et al. Effects of exercise on mitochondrial content and function in aging human skeletal muscle. J Gerontol: Bio Sci 2006; 61:534-540
Wei YH, Lee HC. Oxidative stress, mitochondrial DNA mutation, and impairment of antioxidant enzymes in aging. Exp Biol Med (Maywood). 2002; 227:671-82
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